Advocating for protection, training and better legal protection of the working K9 and service dog. Our Mission: To give all trained and licensed service dogs their deserved status and respect and to reduce the harassment their owners experience.
Friday, April 15, 2016
Tuesday, June 9, 2015
A very belated farewell to a good friend and service dog.
Once again I have to report on the loss of a best friend, companion and assistant.
I do so more than one year after the fact because his loss hit me harder than any other before.
Tys served me for close to seven years with great dedication and loyalty.
He fought degenerative myelopathy for more than one year while still attending to his duties. Fully paralyzed on his hind legs, incontinent and increasingly unable to use his front legs to pull his wheelchair he carried on. A virulent, fast moving cancer finally did him in.
Tys died on September 27, 2013 at the age of 14 years and four months or slightly older.
His loss hit me harder than any other before because Tys was a fighter. He fought for his good life and to remain my companion to his last breath.
Even the vet who had never seen him before and knew nothing about him stated: "He is a fighter, a mighty fighter."
In fact, Tys, my incomparable, extraordinarily loyal and faithful companion and servant did not leave me without doing one last act of service and love.
Knowing that I am all alone, always in mortal danger of a quick demise and that I need assistance, Tys worked the greatest of all miracles: He managed to be reborn or at least to transfer his soul into a look-alike.
Nero is the new Tys.
Only the age difference gives it away. Tys and Nero look so strikingly alike that many of Tys' friends in the park believed that Tys was walking again when they first saw Nero.
No wonder, the mind and soul of Tys resides within Nero. He has taken on Tyson's personality and tasks. He became a trained, state licensed service dog like his two predecessors.
When I follow my three assistants, friends and companions, I do so in the certain knowledge that no man can experience greater blessings than a succession of three dedicated, selfless, loyal and faithful friends beyond the grave.
PJJ
I do so more than one year after the fact because his loss hit me harder than any other before.
Honoring the Life of the perfect
service Dog
Born of an inglorious conception into
the dust, heat, filth and a cluttered backyard in Southern California
he spent the first eight years of his life in an equally filthy,
dirty, hot and uninspiring backyard of a dilapidated house.
Cats, squirrels and rats were his
companions while human contact was a rare exception – except for
more or less infrequent visits by two children of his “family”.
Not a good life for a Boxer that craves
human company and affection!
In fact, that is not a good life for
any dog.
Tys on guard duty.
Then it got even worse. One day the
mother packed children and Boxer in to her car and
headed for the nearest animal shelter. There she surrendered him to
be killed.
The children fought desperately for his
life. To no avail. No help, no compassion other than the sympathy of
bystanders who treated the mother with scorn and contempt when she
tried to explain her action:
“I am sick and tired to see his ugly
black face sitting in my backyard.”
But you put him there, you despicable
piece of human garbage!
I first saw him four hours before his
scheduled execution.
He greeted me with the usual Boxer
exuberance inviting himself to a round of play. Yet, when I looked
straight into his eyes to explore his personality, they changed in an
instant from happy, open and friendly to guarded, reserved and
defensively stubborn.
“Oh, not with me, my friend”, I
told him.
“You just lost your last chance of
redemption”, I added walking away from his kennel.
Something prompted me to look back from
15 to twenty yards away. What I saw was amazing.
A dark paw was protruding as far as
possible from between the bars of his kennel door vigorously waving
up and down. I HAD to return to the death candidate.
“Hey, you just saved your life. But
do not ever do this again”, I warned him walking away a second
time.
And that is how Tyson the unloved,
unwanted, neglected and abandoned Boxer from a filthy backyard from
one of the most run down neighborhoods of the San Fernando Valley
came into my life. He eventually morphed into my loyal, well behaved,
gentle, considered and treasured service dog. His undying dedication
to faithfully serving me in my lonely life lasted until the very last
moments of the six years he gave me the honor of sharing his life and
companionship with me.
Tyson was a wild man at first. He had a
tendency to run away, was scared of everything unfamiliar and shunned
the company of humans for many months. He even stayed away from me
most of the time.
It took the better part of a year to
teach him the basics of living in a house (though he strangely was
house broken from the first moment on) and to rid him of fear of the
unknown.
The stubbornness I first noticed at the
shelter stayed with him causing chaos and disharmony until I
accidentally discovered the key to his heart. After one of his
infamous episodes of stubborn resistance that forced me to “boss”
him unceremoniously into the car, I took his head into my two hands
to explain to him the futility of his resistance. Unexpectedly and in the blinking of an eye it happened: His eyes
glazed over and he smacked his lips in delight.
At that memorable instant Tyson, the
wild man, became Tys, a Boxer that had revealed the way to his heart
and undying dedication. From then on he was easy to train. In fact,
he almost trained himself by diligently observing what pleased me and
by then acting accordingly. It was his way to thank me for giving him
what he had been missing for so many years: Affection and close
contact with humans.
I am disabled for life with a fatal
heart condition that can kill me at any moment. I also live all alone
without relatives nearby. Getting sick and incapacitated even from a
common cold can leave me helpless and unable to summon help. Tys
understood that perfectly well.
When I stricken with a very, very bad
cold that made me fall into coma like sleep over and over again for
two nights and three days, I found Tys sitting straight up right next
to me every time I awoke. His black face had a serious and worried
look on it. He would carefully and closely examine my face, sniffing
my nasal area most deliberately.
Thereafter I taught him the skills
needed to become a licensed service dog. No, not one of the phony
service dogs with a cheap, glaring service dog patch that one can purchase on
the Internet.
He had to do better.
Tys learned everything a real service
dog must know to pass the rigorous test given by the local animal
control service. Tys, grandpa Tys for his was already past his prime,
passed his test with flying colors and became a licensed California
service dog – SM#09. He wore his small official license tag with
pride. It also opened an entirely new world to him that was closed
and unreachable for so long. He enjoyed his new life immensely. It lasted for about five years. Five happy years for a happy dog. He was known as the "happy dog" in our local park - just as his predecessor Windsor.
But it is one of the tenets of life that happiness never can last forever. There seems to be a dark power that eventually will attempt to destroy it. Tys would not have any part of it. In 2012 he showed the first signs of myelopathy. Yet he
continued to run around in the park chasing squirrels even after his hind legs started to fail him.
He stood up like a man to eat when they barely could hold him up. His condition worsened in early 2013. He needed a wheelchair to get around despite expensive treatments, chiropractic care and his undying fighting spirit. He simply refused to give up.
He fought degenerative myelopathy for more than one year while still attending to his duties. Fully paralyzed on his hind legs, incontinent and increasingly unable to use his front legs to pull his wheelchair he carried on. A virulent, fast moving cancer finally did him in.
Tys died on September 27, 2013 at the age of 14 years and four months or slightly older.
His loss hit me harder than any other before because Tys was a fighter. He fought for his good life and to remain my companion to his last breath.
Even the vet who had never seen him before and knew nothing about him stated: "He is a fighter, a mighty fighter."
In fact, Tys, my incomparable, extraordinarily loyal and faithful companion and servant did not leave me without doing one last act of service and love.
Knowing that I am all alone, always in mortal danger of a quick demise and that I need assistance, Tys worked the greatest of all miracles: He managed to be reborn or at least to transfer his soul into a look-alike.
Nero is the new Tys.
It takes a good close look to see the difference.
No wonder, the mind and soul of Tys resides within Nero. He has taken on Tyson's personality and tasks. He became a trained, state licensed service dog like his two predecessors.
When I follow my three assistants, friends and companions, I do so in the certain knowledge that no man can experience greater blessings than a succession of three dedicated, selfless, loyal and faithful friends beyond the grave.
PJJ
Wednesday, June 26, 2013
Degenerative Myelopathy - Statistics To Consider
In the last seventeen years I have had two Boxers with symptoms of myelopathy. One was a confirmed carrier of the mutant gene. The other remains untested. Both were rescued from an animal shelter.
Both lived past 14 years.
That's pretty good for a Boxer and many medium sized breeds. Larger dogs have generally shorter life spans than small dogs.
Since myelopathy does not manifest itself until a dog has reached his senior years, it is almost impossible to determine whether a larger dog you want to rescue is at risk of myelopathy.
It is also highly impractical when you adopt from a shelter because the dogs are not held until test results of a myelopathy test are available. It can take up to 3 months to get the results.
However, the Orthopedic Foundation for Animals has published a list of breed related test results that can give an interested party some statistic hints of the chances for myelopathy carriers in certain breeds. For Boxers, for example, statistics show this picture:
It is therefore reasonable to expect that almost half of all Boxers are most likely to develop myelopathy at a higher age. And about 80 percent of all Boxers carry the mutated gene in one combination or other. Only 17 percent are not at risk. This is confirmed by my own two Boxers. Both developed myelopathy at about 13 years of age.
These statistics show indirectly something else: Popular breeds (that sell well) are very likely to be at risk or carry the mutated gene. The popularity of breeds leads many 'backyard' breeders to produce puppies by random choice of partners. They do not test for the gene before selecting breeding partners. The more popular a breed the higher the percentage of backyard bred dogs with all sorts of genetic disorders.
That is the irresponsible part of backyard breeding. I would apply stringent legal restrictions on it that would 'encourage' breeders to act more responsibly. But that is mainly wishful thinking as matters stand right now.
Even feeble attempts to get a handle on backyard breeding have largely failed. One of the consequences are animal shelters that are overflowing with unwanted dogs and cats. I just come from one that houses more than 370 dogs and close to 140 cats. Yesterday the same shelter had over 411 canine inmates.
Oh, how I love backyard breeders.
If you want to check your favorite dog breed for inherent chances of myelopathy visit http://www.offa.org/stats_dna.html?dnatest=DM
PJJ
Both lived past 14 years.
That's pretty good for a Boxer and many medium sized breeds. Larger dogs have generally shorter life spans than small dogs.
Since myelopathy does not manifest itself until a dog has reached his senior years, it is almost impossible to determine whether a larger dog you want to rescue is at risk of myelopathy.
It is also highly impractical when you adopt from a shelter because the dogs are not held until test results of a myelopathy test are available. It can take up to 3 months to get the results.
However, the Orthopedic Foundation for Animals has published a list of breed related test results that can give an interested party some statistic hints of the chances for myelopathy carriers in certain breeds. For Boxers, for example, statistics show this picture:
| BOXER | ||
|---|---|---|
| AT RISK | 1054 | 45% |
| CARRIER | 880 | 38% |
| CLEAR | 396 | 17% |
| TOTAL TESTED | 2330 | |
It is therefore reasonable to expect that almost half of all Boxers are most likely to develop myelopathy at a higher age. And about 80 percent of all Boxers carry the mutated gene in one combination or other. Only 17 percent are not at risk. This is confirmed by my own two Boxers. Both developed myelopathy at about 13 years of age.
These statistics show indirectly something else: Popular breeds (that sell well) are very likely to be at risk or carry the mutated gene. The popularity of breeds leads many 'backyard' breeders to produce puppies by random choice of partners. They do not test for the gene before selecting breeding partners. The more popular a breed the higher the percentage of backyard bred dogs with all sorts of genetic disorders.
That is the irresponsible part of backyard breeding. I would apply stringent legal restrictions on it that would 'encourage' breeders to act more responsibly. But that is mainly wishful thinking as matters stand right now.
Even feeble attempts to get a handle on backyard breeding have largely failed. One of the consequences are animal shelters that are overflowing with unwanted dogs and cats. I just come from one that houses more than 370 dogs and close to 140 cats. Yesterday the same shelter had over 411 canine inmates.
Oh, how I love backyard breeders.
If you want to check your favorite dog breed for inherent chances of myelopathy visit http://www.offa.org/stats_dna.html?dnatest=DM
PJJ
Tuesday, June 25, 2013
My service dog Tys testing his new Doggon Wheels
Here is a brief video of Tys taking his first steps in his brand new wheelchair. It will take a while before he really gets the hang of it.
But it beats not being able to walk at all.
We have some newer videos showing him interact with other dogs. Will post soon.
Padre
But it beats not being able to walk at all.
We have some newer videos showing him interact with other dogs. Will post soon.
Padre
Monday, June 24, 2013
Dog Breeds Affected By Degenerative Myelopathy
Here is a list of seventeen dog breeds that are prone to get degenerative myelopathy at a later age. Other dogs may acquire the disease as well. The illness is caused by a mutated gene. Not all dogs with the mutated gene will eventually get myelopathy. Some dogs have two of the mutated genes (one from mother and father each), some have only one. The majority of the dogs with two mutated genes will eventually develop myhelopathy - usually at 8 to 10 years of age and above.
This disease is fatal. There is no known cure at this time despite of claims by websites that hawk phony service dog patches and cures for DM.
The best a dog owner can hope for at this time is to slow down the progression of the illness. Most dogs with DM are euthanized at around 8 to 12 months after first diagnosis. Some can persevere longer and enjoy some sort of a 'new' life in a doggie wheelchair.
There is a test for the disease. It costs $ 65.00 and can be ordered from the Orthopedic foundation For Animals (offa.org).
This disease is fatal. There is no known cure at this time despite of claims by websites that hawk phony service dog patches and cures for DM.
The best a dog owner can hope for at this time is to slow down the progression of the illness. Most dogs with DM are euthanized at around 8 to 12 months after first diagnosis. Some can persevere longer and enjoy some sort of a 'new' life in a doggie wheelchair.
There is a test for the disease. It costs $ 65.00 and can be ordered from the Orthopedic foundation For Animals (offa.org).
- American Eskimo Dogs
- Bernese Mountain Dog
- Borzoi
- Boxers
- Cardigan Welsh Corgi
- Chesapeake Bay Retrievers
- German Shepherd Dog
- Golden Retriever
- Great Pyrenees
- Kerry Blue Terriers
- Pembroke Welsh Corgis
- Poodle
- Pug
- Rhodesian Ridgeback
- Shetland Sheepdog
- Soft Coated Wheaten Terriers
- Wire Fox Terrier
The National Academy of Sciences of the United States reported in an article on the research of numerous scientists from a number of famous American universities on the most recent research into the causes of DM and possible treatments. The article was presented by James E. Womack, Texas A&M University under the title
"Genome-wide association analysis reveals a SOD1 mutation in canine degenerative myelopathy that resembles amyotrophic lateral sclerosis.
Canine degenerative myelopathy (DM) is a fatal neurodegenerative disease prevalent in several dog breeds. Typically, the initial progressive upper motor neuron spastic and general proprioceptive ataxia in the pelvic limbs occurs at 8 years of age or older. If euthanasia is delayed, the clinical signs will ascend, causing flaccid tetraparesis and other lower motor neuron signs. DNA samples from 38 DM-affected Pembroke Welsh corgi cases and 17 related clinically normal controls were used for genome-wide association mapping, which produced the strongest associations with markers on CFA31 in a region containing the canine SOD1 gene.
SOD1 was considered a regional candidate gene because mutations in human SOD1
can cause amyotrophic lateral sclerosis (ALS), an adult-onset fatal paralytic neurodegenerative disease with both upper and lower motor neuron involvement. The resequencing of SOD1 in
normal and affected dogs revealed a G to A transition, resulting in an E40K missense mutation. Homozygosity for the A allele was associated with DM in 5 dog breeds: Pembroke Welsh corgi, Boxer, Rhodesian ridgeback, German Shepherd dog, and Chesapeake Bayretriever.
Microscopic examination of spinal cords from affected dogs revealed myelin and axon loss affecting the lateral white matter and neuronal cytoplasmic inclusions that bind anti-super-oxide dismutase 1 antibodies. These inclusions are similar to those seen in spinal cord sections from ALS patients with
SOD1 mutations. Our findings identify canine DM to be the first recognized
spontaneously occurring animal model for ALS . . ."
If you want to know the real facts about the disease and possible remedies and also can handle 'science speak', go to http://www.pnas.org/content/early/2009/02/02/0812297106.full.pdf+html and become an expert in the most up-to-date research into this insidious disease.
PJJ
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